Lumbar Stress Fracture

Lumbar Stress Fracture 364 200 Dr Paul Annett

Lumbar Stress Fractures


Lumbar stress fractures are a common cause of lower back pain in adolescence. Up to 30% of athletes between 11-17 may have lower back pain, and a high index of suspicion for stress fracture should be maintained in this age group.



Lumbar stress fractures occur due to an overload of the pars interarticularis region. The L5 level is most commonly involved. Symptoms occur commonly in adolescence, and generally relate to exercise that involves extension and/or rotation. Classic sports affected include cricket fast bowling or gymnastics, but they may occur in any running or pivoting sport

Stress fractures may occur in structurally normal bone. There are also chronic bony lesions in this area known as ‘pars defects’. A pars defect is not congenital, but develops early in life. It is present in around 5% of the population and is often asymptomatic. It may be aggravated with similar activities that cause acute stress fractures. When the pars defects are bilateral there may be a slippage of one vertebra on another, which is known as a spondylolisthesis.

Fig 1. The single-leg hyper-extension test



Historically the patient will complain of a gradually evolving lower back pain, usually unilateral, which is worsened by their chosen sport and improves with rest. At its worst the pain may be present in daily activities or even at night. Often it will grumble on for many months before a diagnosis is mad

Clinical examination will reveal pain that is worsened by positions of spinal extension. More specifically extension/rotation or single leg hyperextension to the affected side may also reproduce pain. Tenderness may be palpated at the lumbosacral level, 1cm lateral to the midline.



A plain X-ray is the simplest method to investigate the adolescent with back pain. It may show a pars defect or even a spondylolisthesis. Oblique views are more sensitive for pars defects, but increase the radiation dose. Further investigation may be needed to make the diagnosis. This may initially involve a bone scan, which is very sensitive for diagnosing stress fractures and confirms bony activity, and a limited CT scan to stage the lesion as acute or chronic. MRI scanning may be a viable alternative as it avoids radiation, but is not as sensitive as the combined bone scan/CT.



The prognosis for lumbar stress fractures is generally favorable. The treatment will involve complete rest from all sport for anywhere up to 3 months. Bracing has been used historically, but had not been shown to improve outcomes and is reserved only for recalcitrant pain.

The period of rest required will be guided by the stage of the lesion on the CT scan. A Japanese study demonstrated that with rest, union of the fractures occurred in 75% of early lesions, 40% of progressive lesions and 0% of terminal lesions. As such, terminal lesions only require rest until symptoms resolve as healing is unlikely, as opposed to early lesions where healing is the goal and more prolonged rest is needed. Radiological signs of a terminal lesions include sclerosis and widening of the pars defect, bilateral defects and associated spondylolisthesis

Physiotherapy is required to improve lumbar mobility and flexibility. Home exercises to stretch the hamstrings, hip flexors and gluteals, as well as to strengthen core stabilizing muscles should be performed daily. Technique correction is important, especially in cricket bowlers where a ‘mixed action’ of shoulder and hip counter-rotation is a predisposing factor.